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References Dwivedi, Divya, et al

Mechanistically, PCK2-K100la augments its kinase activity and competitively inhibits the Parkin-mediated polyubiquitination of 3-oxoacyl-ACP synthase (OXSM), thereby altering mitochondrial fatty acid synthesis and boosting oxidative phosphorylation and the TCA cycle

"End-of-life targeted degradation of DAF-2 insulin/IGF-1 receptor promotes longevity free from growth-related pathologies"

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Interestingly, NFAT5 could promote NIK ubiquitination via cIAP1/2 E3 ligases, regardless of CaOx-exo presence [57, 58]