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When exposure remains ongoing, inflammatory signaling does not resolve, regardless of supplementation or supportive therapies

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This review incorporates the established model of its pathogenesis that arises from a critical interplay between genetic susceptibilityprimarily driven by HLA class II allelesand environmental triggers, particularly enteroviral infections, through molecular mimicry

Current obstacles to successful local or systemic delivery of peptides include poor absorption, enzymatic degradation, and mucociliary clearance

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