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In vivo , it mitigates bone loss in OVX models, underscoring its potential in vascular-dependent osteogenesis, particularly for the prevention and treatment of postmenopausal OP (Liu et al., 2023)

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Such damage can further lead to mitochondrial dysfunction, triggering apoptosis or necroptosis, which represents a key pathological process in oxidative stress [61]

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Our data show that treatment with strong reducting agents (DTT) or antioxidants (GSH and NAC) abolishes hCC oligomerization in our cell model system

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