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We assume each is Michaelis-Menten and take the K m values from[16]

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In this study, we sought to test the hypothesis that ketamine administration in patients with depression leads to an acute increase in glutamatergic activity in the ACC, as measured by 1 H-fMRS, and that pretreatment with the opioid receptor antagonist naltrexone attenuates this increase

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[7] Fragment 176-191 may potentially engage additional cellular signaling pathways that indirectly promote fat metabolism

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