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Although there is still some debate on the etiology of ASD and the extent to which environmental and genetic variables play a role, only around 25% of cases have a significant genetic link [8,9,10]

In Esm1-CreERTR2 -controlled Flt4 loss-of-function experiments, Flt4 i Esm1 KO embryos ( Flt4 lox/lox Esm1-CreERT2 tg/+ ) were compared to Flt4 i Esm1 HET ( Flt4 lox/+ Esm1-CreERT2 tg/+ ), Flt4 i Esm1 WT ( Flt4 +/+ Esm1-CreERT2 tg/+ ), or control littermates without the Esm1-CreERT2 transgene

J Neurol Sci 120: 213217

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doi: 10.1016/j.fct.2009.01.034