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10.1093/oncolo/oyad008 17 CordesL

The impairment of astrocytic autophagy plasticity due to the loss of LC3B and SQSTM1 function accelerates A aggregation and increases GFAP-positive astrocytes in AD mice, thereby amplifying neuronal damage and memory deficits 13 (Fig

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395 The loss of double-strand repair pathway due to BRCA1 or BRCA2 mutations renders cancer cells more dependent on the PARPs-mediated repair, and more sensitive to PARP inhibition, raising a possibility of a wider application of PARPi in cancer therapy

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This instability can cause excessive cellular proliferation, activation of the DNA damage response, proliferative senescence, and apoptosis (Ogrunc et al., 2014)