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[25] In human cells with mutant WRN, NAD+ repletion restored normal mitophagy, improved fat metabolism, decreased mitochondrial oxidative stress, and enhanced mitochondrial quality

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The cells were collected by centrifugation at 2000 g for 5 min and washed in phosphate buffered saline

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NFE2L2 transactivates multiple genes involved in iron and GSH metabolism, as well as ROS detoxification, thereby limiting oxidative damage (97)

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Posey DJ, McDougle CJ

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This suggests the significance of the hydrophobic nature of the interface in the FOXO4 FHD and p53 TAD interaction

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108 LinnerbauerM.WheelerM

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