We can now begin to decipher which cell types, cellular functions, and genes contribute to smoke-induced injury in normal lung, as well as during COPD exacerbations in individual patients, and thereby, identify common as well as patient-specific disease factors, said Wyss Director Donald Ingber, the Judah Folkman Professor of Vascular Biology at Harvard Medical School and Boston Childrens Hospital and professor of bioengineering at the Harvard John A
J Cell Physiol 233:49354948 Dornan GL, Burke JE (2018) Molecular mechanisms of human disease mediated by oncogenic and primary immunodeficiency mutations in class IA phosphoinositide 3-kinases
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