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-CBT-diol also protected the viability of neuro-2a cells by activating anti-apoptosis protein p-Akt and HAX-1, inhibiting pro-apoptosis protein caspase-3, and regulating the NF-B/VCAM-1/TNF- pathway to resist endothelial inflammation in murine brain-derived endothelial bEND5 cells

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Sharing our heart with a trustworthy friend early in the game before things reached a critical point could have avoided the need for such desperate measures