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Tobacco smoking inhibits expression of proinflammatory cytokines and activation of IL-1R-associated kinase, P38, and NF-kappaB in alveolar macrophages stimulated with TLR2 and TLR4 agonists

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In this regard, to the best of our knowledge, no peer-reviewed work has investigated long-term CB decomposition

At this time, cotinine, the primary proximate metabolite of nicotine, remains the biomarker of choice for assessing secondhand smoke exposure

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