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Some protocols increase to 200 mcg of each after assessing initial response

Nonetheless, the aforementioned animal models, although extremely useful in investigating specific molecular mechanisms in the pathogenesis of ulcerative colitis, do not truly recapitulate disease as observed in patients, as IBD is a multifactorial disease and most clearly is not caused by a single mutation or defect in cellular or molecular pathways

Organoids derived from adenomyosis patients endometrial biopsies demonstrated hormone-responsive behavior, showing cycle phase-specific markers such as Ki67, mucin-1 ( MUC-1 ) and glycogen and maintained cell apicobasal polarity and chromosomal stability [73]

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