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BPC-157 acts predominantly through angiogenic and growth-factor receptor pathways, including VEGFR2 upregulation, FAK-paxillin pathway activation, and eNOS-driven vascular effects

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It promotes lipolysis through growth hormone receptor pathways, which differs from the NNMT inhibition mechanism of 5-amino-1mq

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In general, their protective ability was evidenced by the experimental damage of the skin and gastrointestinal mucosa