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However, analysis of the proportions of various metabolites in plasma over time once again suggested a short half-life and rapid degradation of prototype BPC157

Chang and colleagues (Journal of Applied Physiology, 2011) reported that BPC-157 promotes tendon outgrowth, cell survival, and migration in cultured tenocytes, findings that have been replicated by multiple independent groups and form the basis for its prominence in musculoskeletal-repair research

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