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The deregulation of NO synthesis (both up- and down-regulation, depending on the arsenic concentration) is interesting because this is a signal messenger that promotes vascular relaxation, but during pathological processes, it generates nitrogen reactive species and oxidative stress, damaging the endothelium (138, 139)

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This process produces excess ROS and reactive nitric oxide (RNS), which may lead to an oxidation/antioxidant imbalance [35, 36]

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