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Archived from the original on 26 June 2019

When cigarette smoke extracts (CSE) invade the airway combined with an antigen, TSLP located in epithelial cells can induce the expression of OX40 ligand (OX40L) on DCs, and then promote the differentiation of naive CD4+ T cells into Th2 cells, to drive the production of Th2 proinflammatory cytokines, promote airway inflammation through Th2 cell immune response after that

However, the study notes that people with depression were still less likely to quit successfully than those without, perhaps due to the fear they wont be able to cope without nicotine