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discovered that wogonin could promote oxaliplatin-induced apoptosis by activating phosphorylation of JNK signaling and raising nitrosative stress to accelerate excessive autophagy, thereby synergistically enhancing the chemotherapeutic impact of oxaliplatin on BGC832 cells in vitro ( 154)

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In addition, GSTs may participate in the passive non-catalytic binding of substrates and sequestration, which prevents the binding of xenobiotics to their target proteins [45-48]

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