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However, NADPH can reverse the changes in KA-induced LC3, sequestosome-1 (p62/SQSTM1), and NOX4 protein expression, reduce autophagosome production, and maintain normal neuronal function [122]

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Beyond its central role in apoptotic regulation, the tumor suppressor protein 53 (p53) critically contributes to ferroptosis by suppressing SLC7A11 expression

For clinical translation, non-invasive alternatives (e.g., intranasal NSC-derived exosomes, focused ultrasound-enhanced IV delivery) should be prioritized, as pioneered in recent ASD trials using intranasal routes [37, 38]