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In addition to attacking proteins, lipids, nucleic acids and matrix components, these pro-oxidative agents also serve as intracellular signaling molecules that escalate the inflammatory response in osteoarthritic joints

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Studies have shown that it accelerates the formation of new blood vessels (angiogenesis) and stimulates the migration and proliferation of cells involved in wound repair

For aliphatic GSLs, the formation of 3MSOP (3-methylsulfinylpentyl GSL), 4MTB (4-methylthiobutyl GSL), 7MTH (7-methylsulfinylheptyl GSL), and 8MTO (8-methylthiooctyl GSL) were significantly affected by GSTF11 deficiency (Figure 4C)

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Campbell JD, McDonough JE, Zeskind JE, et al