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BAX regulates dendritic spine development via mitochondrial fusion

It can lead to neuropathy, megaloblastic anemia, and pernicious anemia, [2] [13] causing symptoms including fatigue, paresthesia, depression, cognitive decline, ataxia, and even irreversible nerve damage

Introduction The pharmaceutical industry has witnessed remarkable growth in peptide-based therapeutics over the past several decades

In the treatment of non-unions limited evidence exists [236]

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Based on the available preclinical research, the peptide appears to have several potential mechanisms of action: It may interact with growth factors like VEGF and EGR-1, promoting angiogenesis and aiding in the recovery of various tissues [3, 8]