Moreover, THBS1 can mitigate the menstruation-driven inflammatory response by binding to cell-surface receptors (such as a4b1 integrin) of inflammatory cells, thereby restoring the overall immune homeostasis when proceeding to the next stages of the menstrual cycle [18]
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Role of PI3K-AKT-mTOR pathway as a pro-survival signaling and resistance-mediating mechanism to therapy of prostate cancer
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