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doi: 10.2217/fmb-2021-0173 387 SpecA.PullmanJ.ThompsonG

Journal of Neuroimmunology 205, 148154 [DOI] [PubMed] [Google Scholar] Wang L.W., Tancredi D.J

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The underlying mechanism of sepsis-induced ARDS is very complex, and the general process may be as follows: under the action of injury factors (such as bacterial and viral attacks, traumas, peritonitis and so on), circulating immune cells are activated and inflammatory mediators are overwhelmingly released in the blood, which damages the systemic vascular endothelium, including the capillary endothelium of the lung

Importantly, Dectin-1 can also cooperate with TLRs to fine-tune the antifungal immune response