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The historical perspective Initial clinical findings that the intestinal epithelium of IBD patients is more permeable to tracer molecules known to permeate through the paracellular space 28, 29, 30, 31 led to the hypothesis that an uncontrolled mucosal barrier may help promote IBD pathogenesis

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Oxidative stress reduces the longevity of the ovary by direct cell loss and chronic inflammation

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Minimal reported adverse effects but lacks Phase 3 validation