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Inhibiting ferroptosis via iron chelators, radical-trapping agents, and other interventions demonstrates significant improvements in neuronal survival and synaptic function across diverse preclinical models, establishing ferroptosis targeting as a promising novel therapeutic strategy for AD

53 This reveals a novel BCAA metabolism inhibition mode in the context of KRAS inhibition of BCAT2 ubiquitination degradation, explaining the preference for PDAC amino acid metabolism

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Lim CL, Raju CS, Mahboob T, Kayesth S, Gupta KK, Jain GK, et al

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Clinical Pharmacology and Therapeutics, 85(3), 296304