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While these are highly specialized research applications, they demonstrate the breadth of GHK-Cu bone-related effects

(9) might fail to identify some protease targets, because their high affinity interactions are mostly limited to an 810 residue sequence, and in some cases may be unable to withstand the conditions needed to be isolable and observable by mass spectrometry

10.1038/nm1007 Nat

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These findings are also interesting from an IGF-1R targeting point of view because pharmacological modulations leading to an excess of MDM2 could be a manner to cause a selective IGF-1 inhibition and apoptosis in cancer cells