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Third, because high-affinity sodium-dependent glutamate transport is impaired secondary to energy depletion and the resulting disturbance of ionic gradients (see Chapter 13 ), the energy depletion associated with bilirubin mitochondrial toxicity (see later) adds to the transport failure (or even reversal of transport) and accumulation of extracellular glutamate

Frederickson CJ et al

Rhizodeposition of Organic C by Plant: Mechanisms and Controls

n-3 long-chain PUFA promote antibacterial and inflammation-resolving effects in Mycobacterium tuberculosis -infected C3HeB/FeJ mice, dependent on fatty acid status

3 Watanabe F, Hashizume E, Chan GP, Kamimura A

The NF-B inhibitors attenuate hepatic injury in bile duct ligated rats