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JAMA (1994) 271(23):183643.10.1001/jama.1994.03510470040032 23 WangHBloomOZhangMVishnubhakatJMOmbrellinoMCheJet alHMG-1 as a late mediator of endotoxin lethality in mice
The pathophysiology of PsO has been linked to excess production of TNF-
Endogenus glutathione conjugates: occurrence and biological functions
Under physiological conditions, calcium signals generated by the ER in response to stimulation of acetylcholine (ACh) or CCK receptors are transmitted to mitochondria via a sequence of protein structures: IP 3 R (inositol trisphosphate receptors) channels on the ER, the chaperone GRP75 (75-kDa glucose-regulated protein), the channel VDAC1 (voltage-dependent anion channel 1) on the outer mitochondrial membrane (OMM), and the mitochondrial calcium uniporter (MCU) on the inner mitochondrial membrane (IMM)
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