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Stimulation of these enzymes is one of the most important components of cellular defence mechanisms and the ability of ITC to promote their activation is now understood to be enabled by the Keap1-NF-E2-related factor-2antioxidant responsive element pathway

When cells are under external stress, iPLA2 can hydrolyze oxidized PUFA-PLs to inhibit p53-induced ferroptosis

This peripheral restriction is fundamentally different from compounds like Semaglutide (a GLP-1 agonist that acts centrally to reduce appetite) or ghrelin-modulating agents that directly influence hunger hormones