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Ursolic acid stimulates mTORC1 signaling after resistance exercise in rat skeletal muscle

GPx activity and neuroprotective effects of H 2 O 2 It is generally agreed that ischemic insult facilitates an excessive generation of hydroxyl radicals and, therefore, GPxs have an important role in the defense against H 2 O 2 -induced damage

After incubation at 30 C for 16 h, the subsequent sample preparation and LCQTOF or LCMS (Agilent LCMSD iQ) analysis followed the same methods as described for the Cp1B reaction

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Loss of GPx2 increases apoptosis, mitosis, and GPx1 expression in the intestine of mice