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Thus, it was necessary to expose cells for 48 h to detect the significant change in the expression levels of all Gpx4 isoforms (Casaas-Snchez et al., 4)

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Hypoxia-inhibited DUSP2 expression promotes IL-6/STAT3 signaling in endometriosis

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Subsequent research revealed calreticulins mediating role: LPA (18:1) promotes CRT expression through LPAR1/3-ROS-eIF2 pathways, with CRT knockdown inhibiting VEGF-C induction and in vivo lymphatic vessel density (47)