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To view a copy of this licence, visit About this article Cite this article Luo, G., Niu, M., Li, Y

Ludwig Boltzmann Healthy Science Meeting, Vienna, 2016

Overexpression of disintegrin and metalloproteinase domain-containing protein 9 (ADAM9) in lung cancer reduced LDLR protein stability by promoting its proprotein convertase subtilisin/kexin type 9 (PCSK9)-mediated degradation, leading to decreased cholesterol uptake and triggering cholesterol biosynthesis