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192 Excessive glutamate in the synaptic cleft activates synaptic metabotropic glutamate receptors (mGluRs), which lead to neural excitotoxicity
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These compounds elevate glycine levels in the synaptic cleft, thereby augmenting NMDA receptor activation and potentially enhancing cognitive function and memory (Harada et al
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By inhibiting this enzyme, 5-Amino-1MQ is designed to support healthier metabolic function and fat metabolism
Synaptic functionality of these synapses was demonstrated via the presence of analog-induced spinogenesis, and the colocalization of synaptic markers in newly formed dendritic spines which were coupled with enhanced miniature excitatory postsynaptic currents
