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Overall, these described redox PTMs may explain why C

doi: 10.1515/cclm-2012-0552 72 MorrowGPMacMillanLLamarreSGYoungSKMacFarlaneAJBrosnanMEet al

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Release of immune effectors and/or inflammatory polarization of microglia is a logical suspicion, but there may be a more intriguing mechanism at play as suggested by an interesting series of papers by Lisak et al

Thus, in this clinical manifestation, Leishmania -infected macrophages phenotype would most closely resemble M1-macrophages, despite which parasites survive (211)