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MET is enriched in the post-synaptic density fraction, and HGF administration induces MET phosphorylation and clustering of synaptic proteins such as GluN2B, CAMK-II, and the glutamatergic -amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor GluA1, suggesting a functional connection between glutamatergic synapse and MET signaling (Tyndall and Walikonis, 2006, 2007)

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Tian et al., 2019

Clinical trials have shown that pairing these two agents may result in greater weight loss than either compound alone

A similar phenomenon has been observed in experimental models succinate accumulation activates HIF-1, further enhancing glycolysis and pro-inflammatory factor release to accelerate alveolar destruction (27, 31)