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Mechanistic gaps in pathway crosstalk and causality: Current studies have primarily focused on individual pathways (e.g., Nrf2-mediated antioxidant defense and TGF-/SmadHDAC3-driven fibrosis) but lack insights into the core molecular hubs integrating these networks

Exposure to ROS dissociated Nrf2 from Keap1 and allowed relocation of Nrf2 to the nucleus, where ARE responsive genes became actively transcribed