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Despite higher VLDL-TG secretion in patients with hepatic steatosis compared to healthy individuals, VLDL-apoB100 secretion was unchanged, suggesting that NAFLD patients do not secrete additional, but instead larger and more triglyceride-rich, VLDL particles [110]

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Notably the third one was to outline the possible mechanistic contributors underlying the beneficial effects of these agents, focusing on HMGB1/RAGE/JAK-2/STAT-3, PI3K/AKT/CREB/BDNF/TrkB, AMPK/SIRT-1/m-TOR and PERK/CHOP/Bcl-2 cues