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proposed two mechanisms by which (i) melatonin pretreatment represents an interfering mechanism for Ripk3/PGAM5/CypD axis execution, desensitizing cells to necroptosis triggered by RipK3 activation, PGAM upregulation, and CypD phosphorylation 50 in endothelial cells

10.1007/s10522-015-9578-8 Biogerontology

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doi: 10.1152/jappl.1999.87.4.1381 160 LeeC