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Such neuronal death in a caspase-independent manner was described in stroke, in which NG2 glia produce Bcl-2/E1B-19K-interacting protein 3 (BNIP3), a proapoptotic member of the Bcl-2 family proteins (Li et al., 2013)

This creates massive systemic inflammation

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These in vitro results demonstrated that H2O2 induces Leydig cell senescence, accompanied by FOXO4 nuclear translocation and activation of senescence pathways