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Chelating this zinc signal with the membrane-permeable chelator TPEN effectively blocks the activation of LPS-induced signaling pathways, including the p38 MAPK, extracellular-signal regulated kinases 1/2 (ERK1/2) and NF-B pathways, ultimately inhibiting the release of proinflammatory cytokines such as TNF- [18]

Nephrotoxicity of nonsteroidal anti-inflammatory drugs: physiologic foundations and clinical implications

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Coenzyme Q10 supplementation and oocyte aneuploidy in women undergoing IVF-ICSI treatment

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Virology 242 , 221232 (1998)