Within the context of the Dihexa pathway, which is studied for its ability to support synaptogenesis through HGF/c-Met signaling, having typical BDNF function suggests that the underlying neuroplasticity pathways that support synapse formation and neural network remodeling likely operate within a normal physiological range
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Subsequent the HGF coupling with c-Met as a tyrosine kinas receptor, angiogenesis could be initiated through migration and invasion to adjusting tissues
