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Its activation enhances the conversion of Gln to glutamate and promotes subsequent GSH synthesis, while also leading to ROS accumulation, which inhibits the proliferation of lung cancer cells and promotes apoptosis (46)

MCC950 has not entered clinical use for AP, and broader development of this chemical class has been constrained by safety concerns, particularly hepatotoxicity signals observed during clinical development in other inflammatory settings (91, 92)

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Enhanced redox state and efficiency of glucose oxidation with mir based suppression of maladaptive NADPH-dependent malic enzyme 1 expression in hypertrophied hearts